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AUGUST 2007 Combination
antibiotic therapy in Crohn’s disease (CD). Mycobacterium
avium subspecies paratuberculosis has been suggested as a cause of CD. Selby
and coworkers conducted a study in which 213 patients with active CD were
randomized to receive clarithromycin, rifabutin, clofazimine, or placebo. All
patients were given a 16-week tapering course of prednisolone. Patients in
remission at week 16 continued their study treatment in the maintenance phase of
the trial. A greater proportion of patients receiving antibiotics than receiving
placebo achieved remission at week 16 (66% vs 50%; P = 0.02). By week 52, 39% of antibiotic-treated patients and 56% of
patients in the placebo group experienced at least one relapse (P
= 0.054). At week 104 the relapse rates were 26% and 43% (P
= 0.14) and, during the following year, 59% and 50% (P
= 0.54) for the antibiotic and placebo groups, respectively. These findings show
that, while short-term improvements were observed, antibiotic therapy did not
result in sustained benefit compared with placebo. This does not support a
significant role for M avium
subspecies paratuberculosis in the pathogenesis of CD. (Selby W, et al. Gastroenterol.
2007;132:2313–2319.) CD
medication and birth outcomes. Nørgǻrd and others conducted a
nationwide Danish cohort study to determine the effect of drug treatment on
birth outcomes in 900 children born to women with CD between 1996 and 2004. Data
were obtained from the Danish National Registry of Patients, Birth Registry, and
nationwide prescription database. Preterm births occurred more frequently in
azathioprine (AZA)/6-mercaptopurine (6-MP)– and steroid-exposed women than in
women who did not receive medications (25% and 12.3% vs 6.5%, respectively).
Similarly, congenital abnormalities were more prevalent in AZA/6-MP–exposed
women than in unmedicated women (15.4% vs 5.7%). In an accompanying editorial,
Friedman states that only a prospective pregnancy registry can adequately
differentiate the effects of CD activity and medication use on adverse pregnancy
outcomes. (Nørgǻrd B, et al. Am J Gastroenterol
2007;102:1406–1413; Friedman S. Am J Gastroenterol
2007;102:1414–1416.) Medication
adherence. Bokemeyer and colleagues
assessed adherence to thiopurines in patients with CD treated in a single
gastroenterology outpatient practice. Adherence was evaluated by patient
reporting using a standardized questionnaire and by quantitation of thiopurine
levels in red blood cells. Among 65 patients studied, 6 (9.2%) had metabolite
profiles that indicated nonadherence. The self-assessed questionnaires revealed
nonadherence in 4 (7.1%) of 56 patients. The results of this small study suggest
that adherence to thiopurines among patients with CD is satisfactory (>90%).
In contrast, a larger, longitudinal, population-based study (the Manitoba
Inflammatory Bowel Disease Cohort Study), which included 326 patients, found
that approximately one third of patients with IBD were nonadherent. Predictors
of adherence differed between genders. Low adherence predictors in men included
a diagnosis of ulcerative colitis and active employment. The most significant
predictor of low adherence in women was younger age. (Bokemeyer
B. et al. Aliment Pharmacol Ther 2007;26:217–225; Ediger JP, et al. Am
J Gastroenterol 2007;102:1417–1426.) Push-and-pull
enteroscopy. Pohl and associates
evaluated a new endoscopic tool, push-and-pull enteroscopy, in the treatment of
19 consecutive patients who had CD and symptomatic small bowel strictures. Nine
patients were found at endoscopic assessment to be ineligible for endoscopic
therapy for anatomic reasons or because of severe stenotic inflammation. Among
ten patients with 13 strictures, technical success was achieved in eight and
symptomatic relief lasting for a mean of 10 months was afforded to six. This is
the first report of the use of push-and-pull enteroscopy to manage CD patients
with small bowel strictures. These encouraging results warrant further study in
a larger number of patients. (Pohl J, et al. Eur J Gastroenterol Hepatol
2007;19:529–534.) Infliximab
and risk of latent virus reactivation. Lavagna
and others investigated the effect of infliximab on the reactivation of latent
viruses in 60 patients scheduled for infliximab induction treatment. Blood
samples were obtained prior to infliximab infusions at 0, 2, 6, and 14 weeks.
Polymerase chain reaction (PCR) analyses to detect JC-polyomavirus (JCV),
Epstein-Barr virus (EBV), human herpes virus (HHV)-6, -7, and -8, and
cytomegalovirus (CMV) were performed. All patients were negative for JCV and
HHV-6, -7, and -8 at all time points. EBV and CMV serologies were positive in 59
(98%) and 42 (70%) of 60 patients, respectively. EBV-PCR tests were transiently
positive in 4 patients (7%), and no patients were found to be
CMV-PCR–positive. No clinical viral infections were observed, thus supporting
the safety of short-term infliximab treatment in patients with CD who may have
latent viruses. (Lavagna A, et al. Inflamm Bowel Dis 2007;13:896–902.) Immune-mediated
disease clustering in IBD patients. Weng
and coworkers identified 12,601 patients in a managed care program between 1996
and 2005 who had IBD and a common immune-mediated disorder (asthma, psoriasis,
type 1 diabetes, rheumatoid arthritis [RA], multiple sclerosis [MS], systemic
lupus erythematosus, vitiligo, autoimmune thyroiditis, or chronic
glomerulonephritis). Among patients with IBD, 17% had at least one
immune-mediated disease compared with 10% of patients without IBD. Asthma, RA,
psoriasis, and MS were more commonly associated with IBD. The results of this
study suggest that IBD shares common etiologic factors with other
immune-mediated diseases. (Weng X, et al. Am J Gastroenterol
2007;102:1429–1435.)
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